Background: Tumor necrosis factor-α (TNF-α) plays a role in the pathogenesis of and serves as a biomarker for obstructive sleep apnea (OSA) [1]. Moreover, patients with juvenile idiopathic arthritis (JIA) had a numerically elevated risk of OSA [2-4]. Given TNF-α inhibitors have been widely used to treat JIA, the aim of the present study was [...]